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Mendelian

Mendelian \Men*de"li*an\, prop. a. [See Mendel's law.]

  1. (Biol.) Pert. to Mendel, or to Mendel's law; as, Mendelian inheritance.

  2. (Biol.) Behaving or being in accordance with Mendel's laws of inheritance; -- said of the distribution of inherited characteristics and of traits thus distributed. -- Men*de"li*an*ism, Men*del"ism, n.

Wiktionary

mendelian

a. (alternative form of Mendelian English)

Wikipedia

Usage examples of "mendelian".

It is an excellent example of an inherited defect due to a single Mendelian factor.

The white spotting factor behaves as a Mendelian dominant, and the expectation would be equal numbers of normal and affected children.

It is worth noting that all the well attested Mendelian characters in man are abnormalities, no normal character having yet been proved to be inherited in this manner.

No one who examines the collected pedigrees of families marked by feeble-mindedness, can deny that it does appear at first sight to behave as a unit character, inherited in the typical Mendelian fashion.

Furthermore, most of the feeble-minded cases in institutions, where the Mendelian studies have usually been made, come from families which are themselves of a low grade of mentality.

The first fundamental principle of Mendelism, then, is the existence of relatively constant units, the Mendelian factors, as the basis for transmission of all the traits that go to make up an animal or plant.

These difficulties make Mendelian research in man a very slow and uncertain matter.

It has been pointed out in Chapter V that there are good reasons for doubting that feeble-mindedness is inherited in a simple Mendelian fashion, although it is widely accepted as such.

The similar trait of orthodactyly or symphalangism, which likewise appears to be a good Mendelian dominant, seems to exist in only one family.

Traits like these, which are easily defined and occur very rarely, make up a large part of the cases of probably Mendelian heredity.

They are little more than curiosities, their rarity and abnormal nature depriving them of evolutionary significance other than to demonstrate that Mendelian heredity does operate in man.

Many others, such as baldness, are probably Mendelian but not yet sufficiently supported by evidence.

It has throughout denied or minified Mendelian results, and depended on the treatment of inheritance by a study of correlations.

With the progress of Mendelian research, biometric methods must be supplemented with pedigree studies.

In human heredity, on the other hand, because of the great difficulties attendant upon an application of Mendelian methods, the biometric mode of attack is still the most useful, and has been largely used in the present book.